USP22 Functions as an Oncogenic Driver in Prostate Cancer by Regulating Cell Proliferation and DNA Repair.

Authors

• Jennifer J McCann
• Irina A Vasilevskaya
• Neermala Poudel Neupane
• Ayesha A Shafi
• Christopher McNair
• Emanuela Dylgjeri
• Amy C Mandigo
• Matthew J Schiewer
• Randy S Schrecengost
• Peter Gallagher
• Timothy J Stanek
• Steven B McMahon
• Lisa D Berman-Booty
• William F Ostrander
• Karen E Knudsen

Publication/Presentation Date

2-1-2020

Abstract

Emerging evidence indicates the deubiquitinase USP22 regulates transcriptional activation and modification of target substrates to promote pro-oncogenic phenotypes. Here,

Volume

80

Issue

3

First Page

430

Last Page

443

ISSN

1538-7445

Published In/Presented At

McCann, J. J., Vasilevskaya, I. A., Poudel Neupane, N., Shafi, A. A., McNair, C., Dylgjeri, E., Mandigo, A. C., Schiewer, M. J., Schrecengost, R. S., Gallagher, P., Stanek, T. J., McMahon, S. B., Berman-Booty, L. D., Ostrander, W. F., Jr, & Knudsen, K. E. (2020). USP22 Functions as an Oncogenic Driver in Prostate Cancer by Regulating Cell Proliferation and DNA Repair. Cancer research, 80(3), 430–443. https://doi.org/10.1158/0008-5472.CAN-19-1033

Disciplines

Business Administration, Management, and Operations | Health and Medical Administration | Management Sciences and Quantitative Methods

PubMedID

31740444

Department(s)

Administration and Leadership

Document Type

Article