CpG Type A Induction of an Early Protective Environment in Experimental Multiple Sclerosis.

Authors

• James Crooks
• Marco Gargaro
• Carmine Vacca
• Claudia Volpi
• Matteo Pirro
• Giulia Scalisi
• Antonella Turco
• Rita Romani
• Davide Matino
• Abdolmohamad Rostami
• Paolo Puccetti
• Bruno Gran
• Francesca Fallarino

Publication/Presentation Date

1-1-2017

Abstract

Experimental autoimmune encephalomyelitis (EAE) is an inflammatory, demyelinating disease of the CNS that mimics human multiple sclerosis (MS), and it is thought to be driven by Th1 and Th17 myelin-reactive cells. Although adaptive immunity is clearly pivotal in the pathogenesis of EAE, with an essential role of CD4

Volume

2017

First Page

1380615

Last Page

1380615

ISSN

1466-1861

Published In/Presented At

Crooks, J., Gargaro, M., Vacca, C., Volpi, C., Pirro, M., Scalisi, G., Turco, A., Romani, R., Matino, D., Rostami, A., Puccetti, P., Gran, B., & Fallarino, F. (2017). CpG Type A Induction of an Early Protective Environment in Experimental Multiple Sclerosis. Mediators of inflammation, 2017, 1380615. https://doi.org/10.1155/2017/1380615

Disciplines

Business Administration, Management, and Operations | Health and Medical Administration | Management Sciences and Quantitative Methods

PubMedID

28356656

Department(s)

Administration and Leadership

Document Type

Article