Impaired potassium and magnesium homeostasis in acute tubulo-interstitial nephritis.

Publication/Presentation Date

1-1-1985

Abstract

Although acute tubulo-interstitial nephritis is increasingly recognized as a cause of acute renal failure, little is known about renal tubular function in this disease. We report on two patients with acute tubulo-interstitial nephritis who demonstrated abnormalities in proximal and distal tubular function. The first patient developed hyperkalemia presumably from a potassium secretory defect in the distal nephron. The second patient developed an incomplete Fanconi's syndrome with glycosuria and aminoaciduria and two heretofore unreported complications of acute interstitial nephritis: hypokalemia and hypomagnesemia secondary to urinary losses of these cations. Careful monitoring of renal tubular function is indicated in patients with acute tubulo-interstitial nephritis.

Volume

41

Issue

3

First Page

273

Last Page

278

ISSN

1660-8151

Disciplines

Medicine and Health Sciences

PubMedID

4058627

Department(s)

Department of Medicine

Document Type

Article

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